A. Approaches to and Definitions of the Mechanisms of Action of Herbicides --; I. Applications, Morphological, Physiological, and Biochemical Mechanisms of Herbicides --; II. Basic, Intermediate, and Secondary Plant Metabolism --; III. Supercomplex, Complex, and Defined Systems --; IV. Kinetics and Dose-Response Curves --; V. The Chain of Effects --; VI. Mobility and Selectivity of Herbicides --; References for A --; B. Plant Metabolism --; a Synopsis of Principles --; I. Plant Growth as an Open System --; II. Cellular Organelles and Endomembrane Systems --; References for B --; Photosynthesis --; I. Physiology and Biochemistry of Photosynthesis --; II. Flow of Electrons in Photosynthesis --; III. Inhibition of Photosynthetic Electron Flow --; IV. Unclassified and Quinoid Photosynthesis-Inhibiting Herbicides --; V. Binding at the "Diuron Site" --; VI. Light Excitation of Photosynthetic Pigments and Photosynthetic Oxygen Reduction --; VII. Toxicity after Inhibition of Photosystem II --; VIII. Physiological Effects Induced by Herbicides that Inhibit Photosynthesis --; IX. Herbicides Accepting Electrons from the Photosynthetic Pigment System I --; X. Diphenyl Ether Herbicides --; XI. Herbicides Interfering with Carotenoid Biosynthesis --; D. Energy Conservation --; I. Energy Conservation in Photosynthesis and Respiration --; II. Uncouplers of Oxidative Phosphorylation --; III. Respiratory Uncoupling in the Intact Tissue --; IV. Dichlobenil --; an Inhibitor of Cellulose Biosynthesis --; References for D --; E. Nucleic Acid and Protein Synthesis --; References for E --; F. Microtubules --; I. Microtubules in the Cell Cycle of Higher Plants --; II. Flagella, Cilia, and Other Microtubular Systems --; III. Formation of Microtubules --; IV. Morphological and Cytological Effects Induced by Herbicides and Other Compounds Interfering with Microtubular Systems --; V. Herbicide Interference with Microtubular Systems --; Metabolic Effects of Antimitotic Herbicides --; References for F --; G. Lipid Metabolism --; I. Inhibition of Germination by Thiolcarbamate Herbicides --; II. Interference of Thiolcarbamates with Fatty Acid and Wax Formation --; III. Possible Mode of Herbicidal Action --; IV. Interference of Thiolcarbamate Herbicides with Photosynthetic and Respiratory Systems --; IV. Inhibition of Lipid Synthesis by Herbicides Other than Thiolcarbamates --; References for G --; H. Herbicidal Germination Inhibitors with Unknown Mode of Action --; I. Metabolic Pathways Involved in the Germination and Growth of Seedlings as a Target for Herbicidal Action --; II. Germination-Inhibiting Herbicides with Unknown Primary Biochemical Mode of Action --; References for H --; I. Herbicides with Auxin Activity --; I. Tests for Auxin Activity --; II. Structural Requirements for Auxin Activity --; III. Auxin Interaction with Other Plant Hormones --; IV. Auxins and Nucleic Acid Synthesis --; V. Auxin-Binding Proteins --; VI. Sublethal Effects on Intact Growing Plants --; VII. Effects on Basic Plant Metabolism --; VIII. Selectivity --; IX. Phytotoxicity --; References for I --; K. Auxin-Inhibitor Herbicides --; I. Structural Requirements --; II. Degradation and Selectivity --; III. Inhibition of Auxin-Dependent Systems --; IV. General Metabolic Effects and Phytotoxicity --; References for K --; L. Aromatic Amino Acid Biosynthesis --; I. Primary Mode of Action and Regulatory Responses --; II. Interference of Glyphosate and Glyphosine with Other Metabolic Reactions --; III. Phytotoxicity from Glyphosate --; References for L --; M. Other Herbicides and Mechanisms --; I. Competition with Nitrate --; II. Mefluidide --; References for M.
یادداشتهای مربوط به خلاصه یا چکیده
متن يادداشت
Herbicides are part of modern agricultural production systems and therefore contribute significantly to the economy of agricultural products. At the same time, herbicides are potent and specific inhibitors of plant metabolism and may therefore be used as valuable tools in basic plant physiological research. A well-known example is the photosynthesis-inhibiting herbicide diuron, known to plant physiologists as DCMU, which has become one of the essentials in modern photosynthesis research. Similarly, knowledge in other areas of plant metabolism may be advanced by the use of herbicides as specific inhibitors. This book describes the effects of herbicides on the metabolism of higher plants from the viewpoint of the plant physiologist. The material of this book is therefore, as far as possible, divided into areas of metabolism. This book intends (1) to present the reader with current knowledge and views in the area of herbicide modes of action and (2) to promote the future use of herbicides as metabolic inhibitors in plant physiological research to the advantage of both, the pesticide and the plant sciences. I wish to express my thanks to my colleagues and friends Prof. N. Amrhein, Prof. E. Elstner, Dr. L. Eue, Dr. J. Konze, Dr. K. Liirssen, Dr. W. Oettmeier, Dr. H. Quader, Dr. R.R. Schmidt, Dr. R.H. Shimabukuro, Dr. J. Stetter, Prof.
موضوع (اسم عام یاعبارت اسمی عام)
موضوع مستند نشده
Agriculture.
موضوع مستند نشده
Biochemistry.
موضوع مستند نشده
Life sciences.
رده بندی کنگره
شماره رده
QK753
.
H45
نشانه اثر
B933
1982
نام شخص به منزله سر شناسه - (مسئولیت معنوی درجه اول )