Current Directions in Insulin-Like Growth Factor Research
نام عام مواد
[Book]
نام نخستين پديدآور
edited by Derek Roith, Mohan K. Raizada.
وضعیت نشر و پخش و غیره
محل نشرو پخش و غیره
Boston, MA
نام ناشر، پخش کننده و غيره
Springer US
تاریخ نشرو بخش و غیره
1993
مشخصات ظاهری
نام خاص و کميت اثر
(430 pages)
فروست
عنوان فروست
Advances in experimental medicine and biology, 343.
یادداشتهای مربوط به مندرجات
متن يادداشت
Structure, Expression, and Regulation of the IGF-I Gene --;Differential Regulation of IGF-I Leader Exon Transcription --;Insulin and IGF-I Analogs: Novel Approaches to Improved Insulin Pharmacokinetics --;Analysis of the Interaction of Igf-I Analogs with the IGF-I Receptor and IGF Binding Proteins --;Synthesis and Characterization of IGF-II Analogs: Applications in the Evaluation of IGF Receptor Function and IGF-Independent Actions of IGFBPS --;Towards Identification of a Binding Site on Insulinlike Growth Factor-II for IGF-Binding Proteins --;Transcriptional and Post-Transcriptional Regulation of the Human IGF-II Gene Expression --;Significant Species Differences in Local IGF-I and -II Gene Expression --;Transcriptional Regulation of the Insulin Receptor Gene Promoter --;The Regulation of IGF-I Receptor Gene Expression by Positive and Negative Zinc-Finger Transcription Factors --;Cell Cycle Control by the IGF-1 Receptor and Its Ligands --;The Insulin Receptor Family --;IRR: a Novel Member of the Insulin Receptor Family --;Molecular Properties of Insulin/IGF-1 Hybrid Receptors --;Immunological Studies of Type I IGF Receptors and Insulin Receptors: Haracterisation of Hybrid and Atypical Receptor Subtypes --;Insulin like Growth Factor 1 Receptor Signal Transduction to the Nucleus --;Molecular Cloning of pp120/ ECTO-ATPase, An Endogenous Substrate of the Insulin Receptor Kinase --;The Insulin-like Growth Factor-II/mannose-6-Phosphate Receptor: Structure, Function and Differential Expression --;Parental Imprinting of the Genes for IGF-II and Its Receptor --;Multihormonal Regulation of IGFBP-1 Promoter Activity --;Insulin-like Growth Factor Binding Protein-1: Identification, Purification, and Regulation in Fetal and Adult Life --;Rapid Regulation of Insulin-like Growth Factor Binding Protein-1 Transcription by Insulin In Vito and In Vivo --;IGF Binding Protein-3 and the ACID-labile Subunit: Formation of the Ternary Complex In Vitro and In Vivo --;Role of Post Translational Modifications in Modifying the Biologic Activity of Insulin like Growth Factor Binding Proteins --;Cellular Actions of Insulin-like Growth Factor Binding Protein-3 --;Gene Expression of the IGF Binding Proteins during Post-Implantation Embryogenesis of the Mouse: Comparison with the Expression of IGF-I and -II and Their Receptors in Rodent and Human --;Hormonal Regulation of Insulin-like Growth Factor Binding Protein-1 Expression and the Development of Transgenic Mouse Models to Study IGFBP-1 Function --;Limited Proteolysis of Insulin-like Growth Factor Binding Protein-3 (IGFBP-3): A Physiological Mechanism in the Regulation of IGF Bioavailability --;Effects of Insulin-like Growth Factor 1 (IGF-I) Administrations on Serum IGF Binding Proteins (IGFBPS) in Patients with Growth Hormone Deficiency --;Metabolic Effects of rhIGF-1 in Normal Human Subjects --;IGFS and Muscle Differentiation --;IGF-II in the Pathogenesis of Rhabdomyosarcoma: a Prototype of IGFS Involvement in Human Tumorigenesis --;The Physiology and Pathophysiology of IGF-I in the Kidney --;Regulation of IGFBP-4 and -5 Expression in Rat Granulosa Cells --;Insulin-like Growth Factor (IGF) Binding Protein-1 Is an Antigonadotropin: Evidence that Optimal Follicle-Stimulating Hormone Action in Ovarian Granulosa Cells Is Contingent upon Amplification by Endogenously-Derived IGFS --;Insulin-like Growth Factor-I and Insulin-like Growth Factor Binding Proteins in the Zucker Fatty Rat: a case for Differential Tissue Regulation --;Characterization of the IGF Regulatory System in Bone --;Regulation of IGF Activity in Bone.
یادداشتهای مربوط به خلاصه یا چکیده
متن يادداشت
Initially, this family consisted of ligands (insulin, IGF-I and IGF-m and receptors (the insulin receptor, the type I or IGF-I receptor and the type II or IGF-IYM-6-P receptor).