1. Normal Hepatic Circulation --;Anatomy and Physiology --;I. The hepatic lobule and its three vascular axes --;II. Intrahepatic arterio-portal communications --;III. Normal hepatic circulation --;2. Angiographic Methods --;I. Direct portal venous opacification --;II. Hepatic venous study --;III. Arterioportography --;IV. Computed tomography --;3. The Main Arteriographic Signs of Portal Hypertension and their Hemodynamical Significance --;I. The mesenterico-portal axis opacification --;II. The spleen pattern --;III. Hepatic arterial changes --;4. Presinusoidal Obstructions --;I. Proper angiographic signs --;II. The main causes of prehepatic obstructions --;III. Intrahepatic presinusoidal obstructions --;5. Diffuse Post-Sinusoidal Obstructions --;I. Proper arteriographic signs --;II. Evaluation of the intensity of the obstruction --;III. Etiological diagnosis --;6. Are There Portal Hypertensions Without Obstruction? --;I. Portal hypertension of splenic origin --;II. Portal hypertension and arteriovenous fistula --;7. Contribution of Arterioportography to the Treatment of Portal Hypertension --;I. Checking of surgical porto-caval anastomosis by arterioportography --;II. Treatment of portal hypertension according to hemodynamical changes --;III. Interventional radiology and portal hypertension --;8. Segmental Intrahepatic Obstructions without Portal Hypertension --;I. Segmental reversal of the intrahepatic portal flow: angiographic pattern --;II. Differential diagnosis of segmental reversal of intrahepatic portal flow: arterio-portal fistulae --;III. Etiological diagnosis --;IV. Conclusions --;General conclusions --;References.
SUMMARY OR ABSTRACT
Text of Note
The liver blood flow disorders are usually described under the term 'portal hypertension', which is a well- known syndrome since it has been widely studied for years from a clinical, radiological and therapeutic point of view.