xx, 362 pages : illustrations (some color) ; 24 cm
GENERAL NOTES
Text of Note
Fifth international workshop, April 28-May 1, 2002, Banff, Alberta, Canada.
CONTENTS NOTE
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Modulation of Neuronal Death by the Transcription Factor E2F1 in Experimental Stroke.- Brain Genomic Responses to Ischemic Stroke, Hemorrhage, Seizures, Hypoglycemia and Hypoxia.- Temporal Profile of Gene Induction After Venous Ischemia and Effects of Spreading Depression.- The Role of Protein Phosphatases Type 2C in Neuronal Apoptosis.- Mitochondrial Cytochrome C- and Smac-Dependent Apoptosis in Cerebral Ischemia: Role of Oxidative Signaling.- Erythropoietin: A Beneficial Approach to Neuroprotection in Stroke.- Transgenic Mutants for the Investigation of Molecular Stroke Mechanisms.- Compartmentalization in Focal Cerebral Ischemia.- Modulation of the Post-Ischemic Immune Response Improves Outcome in Focal Cerebral Ischemia: A Role for Lymphocytes in Stroke.- Tumor Necrosis Factor-Alpha (TNF-a) and Ceramide Induce Tolerance to Ischemic and Hypoxic Insults to Brain and Brain Cells Associated with Changes in NFKB function.- Microglial Proliferation and Cell Cycle Protein Upregulation in the Rat Hippocampus Following Forebrain Ischemia.- A Role for Cerebrovascular Endothelium in Ischemia and Reperfusion.- Neural Stem Cells: Developmental Lessons May Yield Therapeutic Insights.- Experimental Neuroprotection: Translation to Human Stroke Trials.- Acid-Sensitive Ion Channels in Brain. New Modulation of Ischemic Injury.- In-Vitro Elucidation of Mechanisms Underlying Cell Swelling and Death of Nerve and Glial Cells.- Endogenous Protection Against Hypoxia/Ischemia in the Brain via Erythropoietin.- Ultrastructural Temporal Profile of the Dying Neuron and Surrounding Astrocytes in the Ischemic Penumbra: Apoptosis or Necrosis.- The Maturation Phenomenon as an Expression of C. & O. Vogt's Theory of Pathoclisis.- Protection by Apomorphine of Dopaminergic Neurons Following Acute Inhibition of Oxidative Metabolism in Rodents.- Marrow Stromal Cells as Restorative Treatment of Neural Injury.- Protein Aggregation, Unfolded Protein Response and Delayed Neuronal Death after Brain Ischemia.- Slow Progression of Neurologic Impairment after Mild Ischemic Insult in Rodents: Relationship to Metabolic and Histologic Changes.- Comparison of Light Transparency Changes of the Cerebral Cortex during Focal Ischemia and Death.- Sphingolipids Metabolism Following Cerebral Ischemia.- Preconditioning of Gerbil Brain Reduces Hippocampal Depolarization Time Following Transient Fore-Brain Ischemia: Relationship to CAI Neuron Injury.- The Expression of MMPs in CNS Injury has Beneficial Roles.- VI Neurogenesis Stem Cell Activation - Repair and Plasticity Postischemic Hypothermia Fails to Improve Outcome after a Striatal Hemorrhage in Rats.- Activation of NG2-Positive Oligodendrocyte Progenitor Cells after Focal Ischemia in Rat Brain.- Postischemic Housing in an Enriched Environment Influences Hippocampal Progenitor Cell Differentiation after Focal Cortical Ischemia.- Estrogen Protects Against Global Ischemia-Induced Neuronal Death and Prevents Activation of Apoptotic Signaling Cascades in the Hippocampal CA1.- Ischemic Preconditioning Prevents Global Ischemia-Induced Downregulation of the AMPA Receptor G1uR2 Subunit in the Hippocampal Cal but does not Bock Caspase-3 Activation nor Protect against GluR2 Antisense Knockdown.- Acute Recovery and Delayed Changes in Tissue Following Transient Cerebral Hypoxia-Ischemia: Impedance, Electron Microscopy and MR Imaging Studies in Rats.- A Selective Thrombin Inhibitor Prevents Thrombin-Induced Neuronal Cell Death and Mild Hypothermia Enhances its Neuroprotective Effects Following Transient Focal Ischemia in Rats.- Zinc Dynamics in the Cerebral Cortex of Adult Mice Following Photothrombotic Stroke.- Persistent Neuroprotection against Focal Cerebral Ischemia Induced by Cortical Spreading Depression in Mice.- Anatomical, Morphological and Behavioral Assessments of Four Different Models of Stroke.- Effects of Endurance Exercise on Recovery Following Focal Ischemic Infarct.- Leukocyte Rolling and Adhesion in Pial Microvessels Following Cerebral Ischemia in the Mouse: Effects of Hypothermia.- Early Hypoxic-Ischemic Changes in Blood Flow, T1 Relaxation Time, Apparent Diffusion Coefficient and Water Content in Neonatal Rat Brain.- Increased Expression of nNos Following Cortical Spreading Depression in Rat Brain J.- Regional Differences on Free Fatty Acid Accumulations between Upper and Lower Frontal Cortex in Rat Focal Ischemia.- Cerebral Ischemia and the Rat Motor Cortex: Time Course of Dysfunction and the Effects of Differential Rehabilitation Paradigms on Functional Recovery.- Rehabilitation Induced Cortical Dysfunction Following Focal Ischemic Infarct.- Behavioral Improvement by Activation of Endogenous Progenitors in the Rat Transient Forebrain Ischemia Model.- 5thlnternational Workshop Maturation Phenomenon in Cerebral Ischemia Round Table Discussion.