Background --;1 Arterial Anatomy of the Eye --;2 Clinical Presentation and Differential Diagnosis of Amaurosis Fugax --;3 Neurologic Mechanisms of Visual Loss --;Pathophysiology --;4 Angiography of Retinal and Choroid Vascular Disease --;5 Visual Aspects of Extracranial Carotid Artery Disease --;6 Occlusion of the Central Retinal Artery --;7 Retinal Cholesterol Emboli and Retinal Stroke --;8 Acute Ischemia of the Optic Nerve --;9 Risk of Cerebrovascular Disease in Patients with Anterior Ischemic Optic Neuropathy --;10 Chronic Ocular Ischemia and Carotid Vascular Disease --;11 The Controversy of Venous Stasis Retinopathy --;12 Role of Vascular Spasm and Ocular Migraine --;13 Nonembolic Sources of Amaurosis Fugax --;Natural History --;14 Natural History of Amaurosis Fugax --;Workup --;15 Noninvasive Vascular Examination in Amaurosis Fugax --;16 Data from CT Scans: The Significance of Silent Cerebral Infarction and Atrophy --;17 Angiography in Amaurosis Fugax --;Therapy --;18 Aspirin Trials in the United States --;19 Antithrombotic Therapy in Patients with Transient Ischemic Attacks or Amaurosis Fugax --;20 The Safety of Carotid Endarterectomy for Amaurosis Fugax --;21 Long-Term Results of Surgical Therapy for Amaurosis Fugax --;Summary --;22 Amaurosis Fugax: A Consensus Statement.
Amaurosis fugax, or "fleeting blindness," has been known as a clinical entity for hundreds of years (1). Transient monocular blindness (TMB) is only one manifestation of a complicated syndrome of ocular, systemic, and cerebral diseases that may include some degree of monocular blindness.