One Introduction to the sequestration hypothesis --;Two The sampling theory of fibrotic arteriosclerosis --;Three Intrusion of atheroma into the most fibrotically thickened intimai sites --;Four Conditions for the intrusion of atheroma in coronary artery --;Five The size of the SMC realm assessed with the help of sampling theory --;Six Biased censoring of low SMC sites by atheroma in coronary artery --;Seven Biased sampling of low SMC sites by atheroma in thoracic aorta --;Eight SMC numbers at varying depths in intima of thoracic aorta --;Nine Histologic appearances of SMC clusters and realms --;Ten Direct imaging of the hypothetical quantity, sequestered lipid --;Eleven Local sequestration of lipid from place to place within an artery --;Twelve Fibroplasia in microscopic renal arteries --;Thirteen Parameters of fibroplasia in renal microvasculature --;Fourteen The course of arterial intimal fibroplasia in aging arteries --;Fifteen The course of fibroplasia per SMC over time in aging arteries --;Sixteen Fibroplasia per SMC in the media of coronary arteries --;Seventeen Influence of arteriolar hyalinization on renovascular fibroplasias --;Eighteen The Hy effect on Ra in widely variable circumstances --;Nineteen Hyalinized renal arterioles and the maleness coronary risk factor --;Twenty Two pathways to atheroma variably linked to renovasculopathies --;Twenty One Age of onset of the sex difference in coronary fibroplasias --;Twenty Two Adrenocortical nodularity in relation to coronary fibroplasias --;Twenty Three Atheroma and intimal fibroplasia in periodontal disease --;Twenty Four Atheroma and intimai fibroplasia in relation to obesity --;Twenty Five Paucity of literature relevant to SMC numbers and the aging risk factor.
The cover of this book summarizes the central features of the sequestration hypothesis: Commonplace appearances seen in human coronary artery, fat stained in paraffin seetions by a new technique explained in Chapter Eleven, are arranged to suggest pathways of evolution toward atheroma.